share email twitter ⋅ join discord whatsapp(2ck)
free120  nbme24  nbme23  nbme22  nbme21  nbme20  nbme19  nbme18  nbme17  nbme16  nbme15  nbme13 
Welcome to zevvyt’s page.
Contributor score: 25


Comments ...

 +1  (nbme22#31)

You dont really need to know the murmur to get the question right, but I'm confused about the murmur. It sounds like Mitral Regurgitation. So why is it in the left sternal border and not the apex?


 +0  (nbme21#27)

Of all the things they can test us on, they're testing our fucking Gadar??

peridot  Don't mean to be an eager beaver but I thought it was pretty cool to get tested on our gaydar! I think that's an important thing to pick up on. As for their answer choices, I'm not always the biggest fan of those since I think there's more than one right way to do something... +1

 +3  (nbme21#37)

A= Caudate ( flanks Lateral Ventricle) B= Internal Capsule, between Thalamus and Lentiform Nucleus( Putamen, GP) C= Thalamus( Flanks 3rd Ventricle D= Temporal Lobe E= Occipital Lobe


 +0  (nbme21#43)

to me, this is a process of elimination question.

Fasciotomy and bosentan don't make sense.

Clopidogrel and aspirin kinda make sense, Except Treatment of DVT is not part of their clinical use. (Clopidogrel 429 and aspirin 475 of FA 2019).

So we're left w Embolectomy.

PS. Thrombolytics and Direct Factor Xa inhibitors are used for DVT

personalpurposes  Yeah I dont think is going for a DVT though, the patient is presenting with "coolness and paleness" in her LE with absence of pulses". This is basically implying an arterial occlusion and thus an embolus in the ARTERIAL system. In a DVT we can say arterial supply is normal so we would feel a warm extremity with normal presentation. Either way I guess at the end of the day you could embolize the DVT clot too. +1
yhm17  And the mention of irregularly irregular rhythms clues you in that a clot was formed in the heart and embolized to the lower extremity arteries. +1




Subcomments ...

submitted by tinydoc(223),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

ishT snieqotu si yevr e,nkyas tub in nsescee iths is twhas nghpp.aein

eTh naaedcclit eoamrlv fo hte PTH lnsgad irugnd ortctmyehoidy ⇒ ↓ PTH

PTH nayrnilo-l: m- e:obn ↑ arvlome of C⁺a² dan hPethpao from -eb noni- ksny:eid ↑ ²a⁺C isrpoteranob nad ↓ ₄³OP⁻ st-bnerio↑-oarp covsnoenir of 25, dyotviHxyrmnia D ot 5,21 tamoyxnHidiyrv D ioCt(lilacr - cetavi rfo)m aiv ↑ tviactiy fo a-1 xeaodysylHr eiynfcedci

rehTrfoee a ↓ TPH dlowu eadl :ot

⇒ ↑ ₄³OP⁻ ⇒ ↓ a²C⁺ ⇒ ↓ 21,5 Hvtardymynxioi D

heT ieqnuost is eansky m(hcu lkie hte erst fo tihs xe)am eecasbu oeesonm hwo sitn fogcusin leraly drah or in a rshu htmgi ikcp eht ntooip C hwere oapptehh is ↑ dan PTH is ↓ TUB ↓ 25 iraimtodvhxyny D

hTis is gowrn as onyl ,152 rnytvyhimiadxo D dowul eb adreecs,de eht novscrseoni reofeb isth era ndoe yb eht ksin n)tlisugh( and rilev.

I yllare ishw yhte ulwod ospt igkanm eht noeiqsstu ionsnfgcu UYERPL for teh ekas fo kganim hmet ougncnf.si Istn ti ohngue taht we aveh to nwko ihts ciodrusiul tumoan of nimo,oaritfn iwthuto vgihna emht nonttyinaeill kiagmn ti rahrde yb ntgpinio yuo to 1 wrnase ieccho ubt iagghcnn a enimut dealti to eakm uoy rwnsea ogwr.n rO singu a aormdn sas naneltuoercm rof a ssdeaie to oidav aknigm ti too pmslie (NGSP = tpalieeri"frov )N"G

tinydoc  I literally got this wrong because I had the font zoomed in and assumed the 1 was on the line above like on uworld when it tries to squish the whole title in the same space x_X +1  
hungrybox  Holy fuck they got me. They boomed me. The fucking NBME boomed me. +2  
graciewacie9  Amen to the PSGN question. They got me on that one. lol +  
msw  the psgn question is pinting to rapidly proliferating glomerulonephritis b/c the pt has developed acute renal failure within days of the insult +  
msw  *pointing +  
snoodle  HOLY GOD MY BRAIN FILLED IN THE 1. i had to read this explanation 4 times to finally see 25-hydroxyvitamin D and not 1,25. F U NBME +1  
avocadotoast  this bs is prob why the question isnt on step 1 anymore +  
zevvyt  so since conversion of 25 --> 1-25 is disrupted , would 25 be high? I know its not an answer choice, just wondering +  


submitted by lsmarshall(393),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

Ari" rostp"led dnuoss ilek rrtysopiare ilasva( or tra)we odprtlse. taniIhnoal fo soxmplaato csysoot ni tac efesc stn'i equti eth se;am not ot ysa I knwo clyexat wtha eht toyssoc era edainlh sa jsut( orscccmoipi ryd atc opop atl?s.)eicpr gonentiIs fo ornodkecedu eamt ot gte het tscsy is ieynrtcla a TRO for .mtsolaxoap

lspomaxaTo as COTRH hsa ditra fo shpuhrodealcy, rlerbaec fconltiaiccasi ,laarcebrrnei()t nda oiesr.hniiitctor hotistrriconeii can eb ni cealtingno VCM ro iopotxamssols. lrvteaPceirniur canoiiaficsclt rae in M.VC taonegniCl VCM luayusl hsa ariehgn l,sos ,uisezrse iplchaeet shra, yrlr“ebbeu nf”uifm sh,ar rthioroiintcsei, nda eiuiertrracplvn ctcaliaoncis.fi

usmile1  also note that toxoplasma can cause the "blueberry muffin" rash (also rubella can as well) +  
raddad  So looking at the CDC website, it looks like "accidental ingestion of oocysts after touching cat feces" is the route you were talking about in the first paragraph, so inhalation of air droplets is wrong inherently. +1  
zevvyt  is his small head common is Toxoplasma? +  


unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

Teh rwnsea is xieleorfhapy uscaeeb the nferfate arc fo eht celmus tetrhcs elfxre ahs ot og rghtuoh eht roalds amri adn sldoar toro .ilnaagg buDm usqoetin, I owk,n tub si’t eht yonl eawnrs atth made e.ssne fI uoy hrtu hte GDR, yuo tno nloy oles rtfnefea tosamic eosynsr ,ebrisf ouy aosl elos het esoyrns ebsdoi nlvidveo ni eth vuaisro eef.xrles

Yuo acn laso tge rpoheelaifxy ofmr nggdaima eth nefefert nesruon thta vtinnaree eth cselum (elik a L,NM) ubt as oyu kown ehtse rea ni eth rtoinrae nhro nad vtrneal ari.m

ankistruggles  Thanks! I agree with you. +  
brethren_md  Great explanation. +  
gonyyong  Agreed - I think I got this by thinking about tabes dorsalis (syphillis) and why it has hyporeflexia is due to dorsal root damage +6  
duat98  I'm confused about why it wouldn't cause muscle atrophy. Isn't that a fever of LMN damage? +4  
charcot_bouchard  Muscle atrophy wont occur because alpha motor neuron is intact. Motor control of Corticospinal tract on this is intact. so no atrophy. u can move shiti/ But remeber muscle spindle that is responsible for INITIATING stretach reflex send Ia fibre to DRG from where it synapse with Alpha motor neuron. if DRG is damage ur muscle is fine but u cant initiate strech reflex. areflexia +3  
zevvyt  DRG you lose DTR +  


submitted by notadoctor(152),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

slaUu atrisiltiten ointumnepis is eht tlihgoiclsao oineintfid fo opthiiacdI ymlprunoa bo.iisfsr We kwon tath tsih epnatit sah ropuymlan srsbifoi sbceeua het iqonsuet satest that teher is sifburo hgineticnk fo het oarelalv st.eap This tionsueq wsa juts getnits ttah ew kenw eth theor sanem fro aPromlnuy os.brFisi

aneurysmclip  Nbme back at it again +24  
pg32  Is it still considered idiopathic pulmonary fibrosis is it appears to have been caused by an atypical pneumonia? +1  
zevvyt  Why not Sarcoidosis? Wouldn't Sarcodosis also be a chronic inflamation with fibrous thickening? +2  
swagcabana  UIP is a better answer. Sarcoid is a leap in logic, usual interstitial pneumonitis is textbook histological definition of idiopathic pulmonary fibrosis. The biopsy has no mention of noncaseating granulomas and the clinical picture is not consistent with an inflammatory process. You have to focus on the better answers, try not to get caught up in the "why nots?" Calling this sarcoidosis is like someone coming in with prototypical asthma and jumping to eosinophilic granulomatous with polyangiitis. Sure its a possibility but its definitely not likely. +6  
mangotango  I picked “diffuse alveolar damage” with Pulmonary Fibrosis in mind but these are actually key words for ARDS :/ +1  
zevvyt  thank you swagcabana! Very good explanation and strategy! +  


submitted by welpdedelp(216),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

tI wsa iabescs, iwhhc is traidettnms noproesn-proe.

welpdedelp  **person-person lol +5  
suckitnbme  NBME loves their scabies +14  
dentist  did you get scabies from "burrows" and "night itching" +  
pg32  My question is where do you get scabies originally? I knew it was transmitted person-to-person, but thought it has to originate somewhere (a pet possibly?) so I went with pets. The internet only seems to say that you get scabies from another person with scabies, so the question remains: where do people contract scabies from? +  
leaf_house  @pg32 , long quote: + "Sarcoptes scabiei mites seek the source of stimuli originating from the host when they are off the host but in close proximity to it. This behavior may facilitate their finding a host if they are dislodged from it and contaminate the host environment. Thus, direct contact with an infested host may not be required for humans and other mammals to become infected with S. scabiei. In the case of human scabies, live mites in bedding, furniture, toys, and clothing can be a source of infection. Sarcoptes scabiei var. hominis have been recovered from laundry bins in a nursing home." + from here: https://parasitesandvectors.biomedcentral.com/articles/10.1186/s13071-017-2234-1 +  
zevvyt  to summarize leafhouse: Fomites +1  
surfacegomd  FA 2020 p.161 "transmission through skin-to-skin contact (most common) or via fomites" +  


submitted by keycompany(296),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

Noreingt nlbcaae is a tmmanureese of itrenpo ealobtsmmi ni eth .body A igevetna renntgio ecalnab acsiinetd mesulc lso,s sa cdraieesn saotnmu fo omani diacs era ebnig edtbeaimzol ot uoepcrd ngyeer. Tihs riaenecss eth nutoam fo rneignto rseceetd morf eth oy.db sBauece the munoat of ronngtie ouy rae itgakn in si lsse atnh het autnom fo nigronte yuo are gsrietecn, uoy aehv a ivgtneae regiotnn nbealca.

hTsi nma si serinou,ahdlm aem,etdsuo ccc,tihae dan sah emboyaiuplnihm.a seeTh illanicc gfdinins pntoi to npirteo namrtnuiotli a(kioshawrK s)iaseeD, hwhci eassuc edmea eud to dereadecs usmer ociotnc rru.sseep wLo tnicoco epesrrus in shit cesa si edu to rtneopi ols,s adn ecneh a ivgatnee igonnetr ncb.laae

drdoom  Nice! +15  
dubywow  I knew your last sentence and suspected Kwashiorkor. It's just everything else I did not know. I have not heard or thought of muscle/protein changes in terms of "nitrogen balance" before... and that's why I got this wrong. Nice explanation! +3  
macrohphage95  I agree with you in first part but i dont think it has any relation to kwashirkor. It is simply due to cachexia which causes muscle destruction through the proteasome pathway .. +3  
zevvyt  also, it says that his albumin is low. +  


unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

I put ocnoitsitanp saeceub I thtouhg teh neicdotima ngebi ieddbrces himgt be C:CB acn meoseno elnpxai why tneitrsa vore ?CBC

seracen  Wouldn't nitrates be a faster acting drug here? That was my take-away anyway. One is more acute, the other for long term maintenance. +4  
suckitnbme  I also believe it's because CCBs have minimal effect on venous beds and would not cause a significant decrease on preload. +2  
beto  decrease of cardiac preload is another word of Venodilation, so Nitrates primarly venodilators. CCB dilate arteria more than veins +  
zevvyt  also, verapamil is the one that causes constipation. But Verampamil is non-dyhydropiridine, so it works more on the heart than the vessels +2  


unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

Teh antptie esuferdf romf nmmueI irombpeotcanhyo.T diaboteintusao sgtaani teh rigyesonlcotp .B3/A2PG

nO blsa, ulylo’ see: ineecras ni g;tcysroeaamkey on eht nuqoitse tmse tyeer’h dcdibesre as “erar tbu el.agr” eyctsorakyMage are nto pesesur.spd

ergogenic22  isolated thrombocytopenia (low platelets) should be highly suggestive of ITP https://www.aafp.org/afp/2012/0315/p612.html +2  
pg32  I agree that in ITP you will see an increase in megakaryocytes, but where did you see that in the stem? Platelets being, "rare but large" doesn't mean megakaryocytes, does it? Also... can anyone explain why she was anxious but alert and had petechiae distal to the blood pressure cuff? +  
meryen13  @pg32, I'm not too sure about the "anxious but alert" but I think they might wanted to mention she is oriented so in case there was no lab values, you would guess that she is not extremely anemic or something. and about the petechia with the cuff and the tooth brushing bleeds, that is a sign of platelet problems because its a superficial bleed. if you saw deep bleeds like joint bleedings, think about coagulation pathway problems (like hemophilia) +2  
zevvyt  "rare" means thrombocytopenia. "Large" means there are megakaryocytes to make up for the thrombocytopenia +2  
lovebug  FA2019, page419 +1  


submitted by cantaloupe5(72),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

otsoglHyi odewhs iegcatuolva srosecni rseveperd( rrheitetcauc fo acoiyaldrm sf)beir ihwt iphlounter inniofttalri wihch htneid thta the IM wsa htwnii 42 huro.s soMt elkyil secua fo htead niwhit frsit 24 oruhs of IM si ih.rrtayahm rMciadoaly tprurue wudol oals be leisbiv on rsgso pnacperaae of hte he,tar cwihh htye isercdedb in the mtse.

bighead478  in FA it shows softening of the myocardium to happen at 3-14 days. Do you think this was overly misleading people (like me) into choosing myocardial rupture? I understand the histo features are consistent with < 24 hours, but the stem should also match this in every detail +11  
sbryant6  Myocardial rupture would not happen until 3-14 days. Since this shows signs of <24 hrs, the answer is arrythmia. +3  
hello  @bighead478 You have to look at the whole picture. Histo shows preserved architecture, which indicates coagulative necrosis -- coagulative necrosis is a histo finding only in the first 24h. The most common causes of MI-related sudden death are: arrythmia > cardiogenic shock (heart pump problem) > rupture. +  
jcmed  I chose the rupture as well due to the timeline. Somebody gave me this advice the other day, NBME classically will give you an entire vignette leading you somewhere, and the what it asks will be something completely different; or in this case will give you a photo of something and will ask about the photo. They do what they want. +4  
athenathefirst  Anyone knows why it's not a cardiogenic shock if it was within 24 hours? +2  
zevvyt  It says "Mottling" which happens in the first day. If it was 3-14 days it would be yellow (p 302 2019). He can be having angina for 3 weeks leading up to an MI. +1  


submitted by mcl(579),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

eolitdD si andvirenet by rxlilaya er,vne hhwic oesmc fmor orost /C.5C6 nctoiAs fo eth doeltid ndlceui oubtacdni of eht rppue eityxm.ret

seagull  I hope everyone memorized every single part of the brachial plexus and all the roots of each, No detail let untouched!!! +26  
mcl  In case anyone else has purged the whole brachial plexus from your memory (like me), this is a great resource linked by another user. https://geekymedics.com/nerve-supply-to-the-upper-limb/ +11  
zevvyt  I thought it was radial since he lost sensation in his thumb. If Radial is C5-T1, wouldn't that be included in C5-C6? +1  
alimd  they force us to know brachial plexus like the holy bible +  


Can't histamine also cause swelling or is it just not involved in the pathology of gout?

zevvyt  yes. Histamine causes vasodilation and increased vessel permeability. But it's not involved in Gout. Gout is more about Neutrophils and Macrophages activating eachother and not really about Mast cells. +2  


submitted by notadoctor(152),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

lUsau litstarntiie netinpmsoiu is hte tgocihlsialo ieinfdinot fo toihcaIdpi oylnmprua s.risoibf eW know ttha shit ntiptae sah aroypnlmu orbifssi abcseeu hte iotnseuq astest htta reeth si bosifur ecnktiighn fo eth varelalo .esatp Thsi inutseoq saw stju stniegt taht ew ewkn hte toerh amnse ofr oPamunylr rFio.sisb

aneurysmclip  Nbme back at it again +24  
pg32  Is it still considered idiopathic pulmonary fibrosis is it appears to have been caused by an atypical pneumonia? +1  
zevvyt  Why not Sarcoidosis? Wouldn't Sarcodosis also be a chronic inflamation with fibrous thickening? +2  
swagcabana  UIP is a better answer. Sarcoid is a leap in logic, usual interstitial pneumonitis is textbook histological definition of idiopathic pulmonary fibrosis. The biopsy has no mention of noncaseating granulomas and the clinical picture is not consistent with an inflammatory process. You have to focus on the better answers, try not to get caught up in the "why nots?" Calling this sarcoidosis is like someone coming in with prototypical asthma and jumping to eosinophilic granulomatous with polyangiitis. Sure its a possibility but its definitely not likely. +6  
mangotango  I picked “diffuse alveolar damage” with Pulmonary Fibrosis in mind but these are actually key words for ARDS :/ +1  
zevvyt  thank you swagcabana! Very good explanation and strategy! +  


submitted by meningitis(502),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

PSL ofnud in roeut amrembne fo mgra ar i⊝cbtae o(htb occci and .)sodr omdesopC of O gtnniae + reoc ccraasiyhlpdeo + ildip A he(t itocx epon)nm.tco

scAvaetti reMsocahpag and ucsedni aNhlFaTp selerea ;g&-t Hnoytpesoni and eevf.r

Pg 331 nooxdt.nEi

zevvyt  and just to note answer b) "induction of histamine release" DOES happen , but it's not the "Initial Event" that the question asks for +  


submitted by cantaloupe5(72),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

eetcrRnru iynkde tensso shdoul cleduin mohphitryiparseryad on uroy flftdenre,aii peuloc ahtt itwh istmraoang dan yreo’u knogiol at NME .1 spoaiLm era sola saodatscie hitw NEM 1.

sympathetikey  Yeah, I probably should have went with that. Just got thrown off, since I know that usually the serum calcium levels for someone with Calcium kidney stones is normal. +  
snoochi95  i understand the link to MEN 1, but why are we checking the calcium level? +  
cmun777  I feel like it's important to get a baseline of where the calcium is at for two reasons: 1. if the patient does indeed have MEN 1 it would be good to know if she has high calcium levels and possible Parathyroid etiology 2. You're putting the patient on a PPI which are known to decrease calcium levels and increase risk of osteoporosis for both these possible factors/concerns it would be good to see where calcium is currently at +5  
zevvyt  Couldn't a Pituatary tumor secrete ACTH, causing high cortisol? +2  
lola915  Patient has symptoms of a gastrinoma (Zollinger-Ellison Syndrome)- patients present with diarrhea, epigastric pain, duodenal and jejunal ulcers. Associated with MEN1 syndrome. +  


submitted by tinydoc(223),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

tfaiscrn dlwou eb a omer ilraeprphe gdewe aehsp

sebcsa odtlwun be anviisev to the ruosnridung raea i .hnkti

maousqsu cell is rmoe acreltlyn oecatdl

nawst' 00%1 sreu btu tthsa eth tsbe aswnre gohhstlu pdiuts ot eivg 0 mposmtsy adn tsuj a rp,tciue nnoigth eilk an ucltaa ilalincc seinoacr

tsl19  Squamous cell is centrally located and has cavitation, which you can see in the pic. Similar to this one: https://webpath.med.utah.edu/LUNGHTML/LUNG068.html +5  
drdoom  ^ linkifying: https://webpath.med.utah.edu/LUNGHTML/LUNG068.html +4  
zevvyt  I also didn't choose infarct cuz i think the lung would have a red infarct. +  


submitted by strugglebus(163),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

no/cHoednhdatMdeyreoo can aedl to deenucpeond-e-y doiav in olng retm es.u ISsNDA yuo lsao aoidv edu to iaarlpt etnvseiefecsfin ni uiecpoahnrt aipn sa wlle sa ecrlu k.rsi 'CsTA era wnnko to rtate ietancouprh iapn eyvr lewl .ei.( d,iaebtse TRA tyhera)p

champagnesupernova3  Drugs for neuropathic pain: TCAs, gabapentin and pregabalin +1  
mangotango  SNRIs +  
mangotango  also SNRIs* +1  
zevvyt  methadone isn't a pain med(even though it's an opiate), it's used for opiate addiction. And hydrocodone is used for "moderate" pain and this person is in "severe" pain. +  


submitted by stepbystep(1),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

sdeo some idmn ngniixalep ywh isht 'sint a erta ni the siiacct e?nrev

sugaplum  It is a very thick nerve, so I think it is hard to tear without physically cutting it. Also if it tore you would have tibial and common fibular nerve symptoms as well. You would see sensory numbness and tingling along the dermatome also the mechanism of injury is focused on spine so a disc rupture is more likely +1  
zevvyt  I got this question wrong but I really like because it helped me get past a confusion I hadon this subject. If it were a tear, you'd see the loss of motor function that sugaplum was taling about(FA 444 2019). But if it's a herniation, like in this case, you see Radiculopathy/Sciatica symptoms that are on 446. +2  


submitted by hayayah(1056),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

In rrdoe rfo a gdru to eb caeredl yb het ,edknyi it muts iftrs eb efidterl in the uoglrm.lei usrDg wtih a ghhi DV hvea ermo of eth gudr ni teh teissu hatt era not lbalivaea ot frtldiee by the keinyd. sDrgu hitw hihg nrptieo dinnibg tow'n be idfetrle iet.erh oS you anwt a rudg whti owl dV and wlo ignnbid fi ouy atnw ti caeldre avi teh ndkeiys nad run.ie

zevvyt  But a low VD corresponds to high Plasma Binding Concentration(FA 233, 2019). That's my main confusion with this question. +2  
kevin  If it's high plasma binding, then it's low Vd. But, low VD doesn't necessarily require high plasma binding. Low Vd can simply be due to it being a large polar molecule +  


submitted by hayayah(1056),
unscramble the site ⋅ remove ads ⋅ become a member ($39/month)

eAcut iteatsiirntl rlnae f.iltamnnimao Pruyai acylslacl(is sh)oilnpeios dna teimzaao rcoucgnri earft aosintinrtdima fo gusdr ttah atc sa ,hatpnes ciinudng eietipntsrysihvy ,(ge tiucirsd,e ND,SAIs plneilcnii evivtiedras, topnro mpup snr,hiiibot iimnfra,p nqnl,uioeso aio)smenfsuld.

hungrybox  But how is a 2-year history acute? +4  
jinzo  there is also " Chronic interstitial disease " +4  
targetmle  i got it wrong because there wasnt rash, also there was proteinuria, doesnt it indicate glomerular involvement? +2  
zevvyt  Got it wrong too cuz of that. But there can be proteinuria in nephritis, just not as much as in nephrotic syndrome. I guess that's confusing cuz this type of nephritis isn't grouped with the other nephritic conditions. +1  
lovebug  FA 2019, Page 591. +