Anyone else got thrown off by the wording of "......each kidney" when the question is referring to A (singular) horseshoe kidney?
I got this wrong I thought Decreased Myosin Light Chain Phosphatase activity would play a factor from the long leg cast immobilization for 6 months. Not being able to use the leg would cause atrophy. But I was wrong.
I got this wrong, thinking it was myositis ossificans due to the history (six days ago he was evaluated for twisted knee at a game with improvement of sxs until one day ago).. guess i should have payed more attention to his PMH
I get that the bruit = Renovascular disease. But why is it not hyperaldosteronism when there is hypertension + hypokalemia + elevated bicarb
I think @beastaran1 has the correct explanation but I just want to say his kidney is "abnormally small and nonfunctional" which means that it is not being used. When the body doesn't use something (like a muscle) it atrophies. So tubular atrophy makes sense.
Atrophy = gradual decline in effectiveness or vigor due to underuse or neglect.
When a question seems wildly complicated and you have no idea what's going on, take a deep breath and explain it to yourself like you're five.
She met the Centor criteria for empiric antibiotics, why was is culture?
Always best to discuss pregnancy prevention prior to pt participating in sex, if not it will be too late
Dementia is a risk factor for aspiration
Just as another POV, you might have seen well-demarcated and thought erysipelas..but remember that is not bullous. Plus, everything else fell more in line with Type 4 HSN.
Ca Gluconate must be given immediately if K > 6.5; think of it as "no point in hydrating or pushing K into cells with insulin if they will die rn of an arrhythmia"
Ca will stabilize immediately, which is why its initial tx.
LOW FLOW (HYPOTENSION) ...> LOW SV...> ^SYMPATETIC
Normal hormonal, male sperm study and normal semen: still can’t conceive then give TYR (???) with clomiphene, that is antagonist at GnRH receptor at hypothalamus, block estrogen mediated inhibition of GnRH hence increase release of LH surge Bromocriptine - would be helpful if the pt has infertility due to INCREASE LPEDS CTIRAON (??? I am assuming its PROLACTIN) - is stopped in US due to clear cell carcinoma in young female born to term. Ethinyl estradiol - bind estrogen & GnRH; LH surge: no use. (Inc risk of endometrial carcinoma) Medroxyprogesterone - is progesterin bind progesterone eonis(oeeccn.rprpmot..t. (???) of morning after pill)
If anyone else is on the dumdum boat and didn’t know what a paranganglioma was: a neuroendocrine tumor that begins in nerve cells, closely related to a pheochromocytoma
Aminoglycosides are synergistic with beta lactam antibiotics (and penecillin is a type of beta lactam antibiotics). The question is saying "what will make this work faster?" basically
why can't this be chronic bacterial prostatis?...ughh the gram stain and leukocytes on UA threw me off
Motor + Sensory + rapid progress :: may be R/O Acute inflammatory demyelinating polyradiculopathy
i think this was tested in a similar way in another question in this form. the patients have a right to know or not know their resuts.
MM will be induced a AL(primary amyloidosis).