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Welcome to graciewacie9’s page.
Contributor score: 7


Comments ...

 +1  (nbme24#19)

I chose residual volume, but looking back, here's why its not (correct me if im wrong):

Restrictive lung disease is an issue with inspiring enough O2, not a problem with expiration (which is in obstructive lung disease like emphysema from floppy alveoli)

Also, a factor that influences A-a gradient is thickened membranes (decreased ability to diffuse across)

I think "Alveolar-arteriole PO2 difference" threw me off bc they didnt use the exact term of A-a gradient and I wasnt able to deduce that they're the same thing or felt like i was being tricked


 +0  (nbme24#19)

I chose residual volume, but looking back, here's why its not (correct me if im wrong):

Restrictive lung disease is an issue with inspiring enough O2, not a problem with expiration (which is in obstructive lung disease like emphysema from floppy alveoli)

Also, a factor that influences A-a gradient is thickened membranes (decreased ability to diffuse across)

I think "Alveolar-arteriole PO2 difference" threw me off bc they didnt use the exact term of A-a gradient and I wasnt able to deduce that they're the same thing or felt like i was being tricked





Subcomments ...

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sMta llsce ltur,eaganed rupdongic ehinsamti hwihc actstatr p.nlshoseoii heT eyalr seagt fo an lerlciag aoiencrt is atsm lcle deid,amet tbu teh elat teags (ngdiuncli msucu inuoot)crdp is dmdtaeie yb ehosisiplno.

atstillisafraud  Thanks for a good answer. This question made me feel like I was taking T21 pills +15  
medguru2295  Thank you- I was really thinking this question had 2 correct answers... of course my dumbass picked Mast cells. +4  
ajss  where do i find this info?? +  
paperbackwriter  @ajss pg 112 of first aid 2019, under type I hypersensitivity. Immediate --> mast cells releasing histamine and tryptase, late--> eosinophils and leukotrienes recruited via chemokines +2  
graciewacie9  Wow, i missed the fact that the question is asking for the RESULT of the reaction, NOT the cause of the reaction. Mast cells cause the initial reaction, eosinophils would be the result of the eosinophils. *facepalm +6  
greentea733  @graciewacie9 SAME UGH +  
lba9587  Pathophys (as far as I understand it)...Mast cell degranulates, thus the phospholipid bilayer et. Al are left behind and needs to be degraded. Who comes in? Our good friend eosinophils, as they contain Major Basic Protein (responsible from breakdown of expired mast cell). Note, you can tie this in to the delayed Leukotriene effects of an allergic rxn, as the bilayer is also broken down by arach. Acid. (See this link to support my credibility https://images.app.goo.gl/3cUF3ZVc7qy8uxAi9) +  
mutteringly  who else looked up what T21 pills were +2  


submitted by tinydoc(223),
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tinydoc  I literally got this wrong because I had the font zoomed in and assumed the 1 was on the line above like on uworld when it tries to squish the whole title in the same space x_X +1  
hungrybox  Holy fuck they got me. They boomed me. The fucking NBME boomed me. +2  
graciewacie9  Amen to the PSGN question. They got me on that one. lol +  
msw  the psgn question is pinting to rapidly proliferating glomerulonephritis b/c the pt has developed acute renal failure within days of the insult +  
msw  *pointing +  
snoodle  HOLY GOD MY BRAIN FILLED IN THE 1. i had to read this explanation 4 times to finally see 25-hydroxyvitamin D and not 1,25. F U NBME +1  
avocadotoast  this bs is prob why the question isnt on step 1 anymore +  
zevvyt  so since conversion of 25 --> 1-25 is disrupted , would 25 be high? I know its not an answer choice, just wondering +