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Comments ...

 +0  (nbme24#8)
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I uotthhg fo it as, a eaBt soaitng owldu uecsa adn,iloiatsvo dan lrfexe rhit,aydcaac rhe lepsu is elardya at ,100 so it wloud mujp hrhg.ie heS uldco gte pitntpoiasla and mtesr.or obbaPlyr het etasl kiylel tlxonapaeni tuo of het ters othuh.g


 +3  (nbme23#48)
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I tsuj zdelriea thta the aernsw I lesdcete ovud'ewl neeb sedoencrdi ipseasv sa:egiergsv ve"Ha you ioednrcdse ltiakgn to a fihat ahrlee ot ese fi erhte rae ayn eothr easersum you shdlou "?rty

omla


 +9  (nbme23#16)
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aaptnnxEisol ofr tihs rae oto mdolcp.cteia kinTh fo it kile isht:

voue'Y ogt a iecpe of tetudma DAN atth is eabl to eb edigetds by a ictsorniret ndcoesna,leue that mensa eht NAD was lyatlrpirinascnot ailbaalev to bnegi .thiw AKA it was not ehlyadett,m ceseabu as ew nwok, tylihmontea = moohantetrhiecr wcihh is rlrpcntanialiytos vein.tica hatt sanem lamytseeh was aeumtdt

lOny toher iuslbplae sawern swa ,DNase nda if it saw tumetda ti oluwd be aeciin,tv otn ecevaroivt.

djeffs1  I mean sure... but this is a prokaryote... +

 +26  (nbme20#20)
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Yuo anc scosr tou eht opt etreh erwsan cceois,h CBA,., ouY otwn eb girnrebsbao gayhtnni in hte TPC in naniocs'f senoymr.d ikLoong at imy,ahakoepl aptoy,ameihrn and yoathppmhohpiaes onw. mkHoaieaply 'tnac eb tecrroc ecaeubs veen houhgt musosipta si oslt ti lliw eb eorasbdbre ta eht etalr itkch neicadsng loop nda fi atht dsnote kema ense,s hte ydob lilw jsaudt fro wlo rmseu ausmptsoi tub cgintiatav the /HK++ pupm on esllc. tI 'tsin artniyhpoame becasue ta teh nelgolctic cutd nclrppiai selc,l tparrnoosbei ilwl o.ccru Thsi vsleae asthoayhpopphmei as teh retorcc adn ynlo warsne heo.icc

imgdoc  by* +2
larascon  Excellent explanation, thank you ! +1
waterloo  It's worth mentioning, that 25% of Na is reabs in the thicc ascending limb. 67% of filtered Na is reabsorbed in the PCT, whereas 85% of phosphate reabsorbed in the PCT. So pt more likely to show Hypophos. +
coconut  UWorld QID:7626 says lab results in people with Fanconi syndrome will show hypokalemia +
j44n  coconut i thought that too but then i remebred fanconi is associated with hypophosphatemic rickets/ VIT D resistant rickets and this little dude is going to be growing because he's 5 and thats gonna increase the demand for phosphate in his body +

 +1  (nbme20#43)
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I kwno tsi' neitgmpt to nktih hatt this amy be dnengAor senytitiinvis sorndyem tbu tis nt,o for ti ot be ISA eerht eeeddn to be na nscreiae ni ee.sotoentrst In hsti sace ti wsa in the lanmor egran fro a .flmaee woN taht ew nwok hte ernsop si fienidtyel meafle &-;tg arenulliM enesigsa ocseebm eth sbet re.wnsa


 +18  (nbme21#33)
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I hotguth otaub it leki sith: Ptantie is igvanh pgniehocsyc ED - tis all ni hsi d,aeh iiodlb is mlonra adn aunnclort cioenrest era mlra.no

If ish nesotsoeteTr lelev was owl - iodLbi esrea.ddec If his ersvnuo meytss rmpitpthaasac)hy/m(teayticeps was gaddema - rcnalotun iosnrcete e.ddraeces

eoHp tish elh.sp

sars  Idk about this. My thoughts: -libido is a product of testosterone and emotions (psychogenic) -tumescence is a product of pelvic splanchnics S2-S4 (parasympathetic) -patient's issues are 1 month old (aka recent) and his wife died 2 years ago & testosterone is normal (therefore libido normal) -patient can jack off, therefore penile stimulation reflex arc is intact and pelvic splanchnics are working (nocturnal tumescence normal) +2

 +6  (nbme21#12)
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Teh inillcac norsatietepn si atht of leorw inabmodla a,pin fveer, dna cish.ll Tshi alone aedm me nikht it saw na ftniymalraom spc.rseo oAls eth oeuniqst syas rehet rae 3 rapseeta polyro edtimdiel erogins fo oawrnr numle. sA far as ulecvirtea tcoslii si done,rccne rethe rea no spik ses,ilon ti si csnutoouin werevher ti s.i iThs dlecupo hiwt hte tohsiyr fo sionatpcnito keams ecidirtiivsult the ebts rnewsa cc.ieoh

lovebug  I know it's silly Q. but why not C) Granulomatous colitis? lol. thank you! +1
trazobone  Granulomatous colitis is Chron’s, which also doesn’t fit bc it presents commonly in the terminal ileum and does not usually present with fever as it is an inflammatory pathology rather an infectious one. While it does present with string sign (“narrowing of the lumen”), anything can cause a narrowing of the lumen, so this doesn’t necessarily mean that it’s string sign +1

 +0  (nbme21#15)
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iIeollcoc imro)s(in/ericsteyf eecmteinsr ,)(arptlo adn ecsnlip (tropfleatl)/ rnlae scset(ymi) rea the loyn wto ietotcrsmopsy hsutn sn.iopot Of cwhih iplencs nda ltfe ranel is hte BSTE tpooni of eth tow ujts sbeda on hrtei rxpyimiot to het gaeaoslhep sc.aeivr

msyrett  ileocolic vein is a portal vein, it empties into the Superior Mesenteric Vein +1

 +2  (nbme21#33)
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uQenoist enstepsr na spuendsuremoispm ptnieat ihtw hrsa evor eht rwelo kbca hihcw si rcui.pitr isclsVee alms(l dulfi ldleif slitesr)b era restpne with cedreesu/trtlcadu soeisnl asol dfnou ni thta aa.re hsTi si ariallcev sozetr itofci:enn lutmplei socpr of olnsise ni ovarusi satsge frmo sveiscle ot uscsrt.


 +7  (nbme21#4)
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iTsh soueqint si gsaikn botau hte iniulns rtoceper setoyrni niekas apthwya. So kate it frmo ,erthe

  1. hTe trpyhoooilahpsn of eht SRI iactevsat a gslian astircnunotd sdecaca taht aesld ot eht notaivtica fo rtoeh iesnaks sa ewll sa irnotiscantrp frcsato ttah emedtai het aeilucltalrrn fcseeft of nln.isiu pmtr/oclsyNuecCliaa tshitlung - sye re(e)isbrvle

  2. o/neernheeinrTiS skensai rae lsao wnnok ot recedu hte ctiivtya of isn.nuli - eys )si(eevblerr

  3. tibUinqui - etaemdid prsseotilyo - on no(t lier)es,bevr and losa nsnuili mylcableiotla nisesreac pnierto tsnhyessi os ti tnd'eso tahmc twah lnniusi deso sywny.aa

ccroetr me if mI' gwro.n

usmle11a  i think youre correct cause ubiquitin mediated proteolyis is an irreversible step. +1

 +0  (nbme21#48)
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I kinht the anrsew saw unconsses cueabes eht u"at"rssoreg aak fylaim eerbmms teanhv dtaeeleribd y.et fI isht wsa rafet tirhe tbidnaeoreil nda tyeh raeesigd, hnte it osge ospuse &;t-g lutda dnrilhce g&;t- strapen -g;t& singilsb .etc The qotnesui tenods' tinenmo yna etms,sagedeinr ecneh htye need to eddeic uicualmelvty dan meka a cdoieni.s

Ttah saw tjus my atek no siht iseoq.ntu


 +6  (nbme21#11)
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Blycaslia isth seutiqon jtsu kass ah"wt is rep"wo adn saks oyu to pnleiax ttah 8%0 .opewr oewrP is 1 - baet y(tpe 2 r),rero asyiablcl wnhe a iefecrendf xtisse nda the ulnl pihyesthos is dtjcreee si pewor. So if teh xR tctsdee a anem efcnidfeer fo 04. ni shmaat in het ptnsteia ni eth tamenetrt pu,rgo tnhe ahtt taad salfl in eht 08% orpew nge,ar nad tis nscfeaicinig is 9%5 0.t&;,l5p(0) P vaule si sjtu teh opbtiblriay fo mnthsoeig hgenpniap by ahc,nec os yuo ahtw ihts ot be less taht %5 so areehwvt ouy osdvereb sint' ulihtlbs.

I ehop hist l,eedph and oetrcrc em if m'I n.ogwr

yssya1992  I have a question : whats the relation then between power and P value ? +
privatejoker  This one took me a minute and was during the last block so my brain was already fried. But my reasoning was that, as stated above, since it gave you power, it is basically just a long-winded way of asking what Power is, and how this relates to p-value. P-value is the odds that the finding was due to chance alone. Obviously a p-value set to <0.05 implies a greater than 95% chance that the finding is legit. Since the power is said to be 80%, this means that there is an 80% chance that the study finding is legit, at least insomuch that it met the pre-set criteria of being 95% non-chance related. +
sahusema  80% chance (power) the study correctly identifies the existence of an association in reality. If an association is determined to exist, >95% chance the study and reality agree with each other (p<.05) +1

 +27  (nbme22#37)
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I ikhtn tsi hte tigrh FLM eraa( C), ont the trghi bdcnasue snuleuc atth is seoendli no het rcoss .tecsnio If eht uabdecsn elsucnu reew siedonle esh tdwnu'lo hvea iuoadnbct ni ehr flet e.ye Teh MFL desmitea csros latk etneebw eht ucdbaens ulnusec no thob ssdei to teh MLF on teh peostpoi deis (2 abeudsnc niule,c 2 MFL oen no hcae de).is nI ehr ce,as reh ghtri LFM stwan' gotniuinncf cneeh wyh hes wsa zgnagi fetl ubt rhe thrig eddmil utcsre wt'san nigantccto to aidetme featwrld g.zae

ishT ctiuper heslp: cstpeo5mbn.m-n/0hwnt1opgwhpt/ha//1oc_42/l-Fhp/t:-/te.pwo20thdm9twujau

imgdoc  *medial rectus +

 +20  (nbme22#1)
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I hkitn tloa of leppeo htigm evah evor pazdhiemes how mnoapittr NPA nad BNP ayrlel ea,r sye it is mriapottn ot knwo hseet stdeepip tge cdsteeer by eht ltiitn/lvrrraaaecu orudmcaimy duirgn treah ale.uifr oweHvre trhie raevllo sfeenteeivsfc ni arntigte aehrt ureailf si h,lzic a eocppertr tdol em thta if NAP and PBN erwe so sfuleu in eiusanrtirs etnh hwy do we egiv ird?icuste tIs' useaebc RAAS orvpswreeo hits teysms chnee cagnusi inevgtea tfceefs nda the dneslse pool fo haret ule.ifra KAA why ew give AEC ibrtn.hsiio

gKnniow ttah ANP gets eiuetndrlaz by hte ARSA ,stemsy ew cna thfis oru soufc kcab to tehar reiaflu ni tish epiatn,t hrwee cdarcia uputot is ere,cdedsa igdelan to HAD risecento nad lfanliy ialtndouil hoapmri.ayent

almondbreeze  a concept continuously emphasized by uw, but I get always wrong :'( +2
almondbreeze  good work done! +
raffff  why does the body make anp at all since its so useless +3
makinallkindzofgainz  @raffff - at least BNP gives us a good marker for heart failure exacerbations :) thanks body! +
mannan  Yeah it's important clinically because it has a high sensitivity (if negative, rule out) for Heart failure. +
alimd  At the same time ANP inhibits renin release? +




Subcomments ...

submitted by imgdoc(135),
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uYo nca srocs tuo eht top rteeh rsnewa eh,socic CB,,.A ouY nwot be erbbgaoinsr athiynng in the CTP ni ifn'nocsa ro.eydmsn kgoLnoi at palkymhieo,a h,omrpayeitna dan phooithphpsamaye onw. yaepkmoalHi nta'c be rcotcre beueacs eevn tghhou smsauoitp is otls it lwli eb eersdaobrb at teh teral ichkt nednsiacg polo dan if htat ostedn kaem esne,s teh ydbo wlli jatuds rof wlo umres smatsioup utb cgtvaatnii hte +/K+H mpup on cllse. It tin's pterhaminoya cesueab at het ccelotginl tudc pcnralipi lc,sel rtbpaooneisr lliw croc.u hTsi valsee aopyhtehmhppisao sa the tceorcr nad yoln wrensa ehoci.c

imgdoc  by* +2  
larascon  Excellent explanation, thank you ! +1  
waterloo  It's worth mentioning, that 25% of Na is reabs in the thicc ascending limb. 67% of filtered Na is reabsorbed in the PCT, whereas 85% of phosphate reabsorbed in the PCT. So pt more likely to show Hypophos. +  
coconut  UWorld QID:7626 says lab results in people with Fanconi syndrome will show hypokalemia +  
j44n  coconut i thought that too but then i remebred fanconi is associated with hypophosphatemic rickets/ VIT D resistant rickets and this little dude is going to be growing because he's 5 and thats gonna increase the demand for phosphate in his body +  


submitted by celeste(83),
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eiWhl teh imifeelt rski ni hte anrgele lontuoipap si jstu woble ,1% it is %6.5 in dsieeetfg-rr aitversle of tatniesp nad it rises to ermo ahnt 0%4 in goyomozinct ntswi fo detcafef pe.olep Anziynagl sliccas eusdsit of teh igtcnsee of hraiispezcnoh dnoe sa rylea sa ni 3s019, escrhiF ncoedclus atht a adcronnoecc tera rof hicysssop of batou 50% in gncyomtoozi tnwsi smees ot eb a lctiiaers tsaeti,me ihchw is tnsicagifilny hrgihe hant ttha ni dtigziyoc tnwsi of bouta 1–019% (.inl5/gn#3le6Mbpr6chic4a9.tm/s3rnC/ome2c./fvPi)

imnotarobotbut  How is one supposed to know this before having read this article? +33  
imgdoc  This question falls under the either you know it or you dont category. It isnt in FA or Uworld +  
jaxx  So why would these A-holes put it on there as if prepping for this exam isn't stressful enough :-| +7  
doodimoodi  Lol just why seriously +2  
champagnesupernova3  This was mentioned in the Kaplan behavioral videos +  
usmlecrasher  and there's so much unnecessarily BS instead of real questions +1  
j44n  I'm just glad we're seeing this garbage now instead of having an aneurysm in the prometric center +3  


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It iads it aws aflta to aslem in retou, adn hte isetunqo dekas btoua ievl onbr fgin.pfors Sneic het elasm atnre’ ngbei nrbo in teh srift eaplc, I aisd 5%0 saflmee and 0% se.mla

hungrybox  fuck i got baited +32  
jcrll  "live-born offspring" ← baited +25  
sympathetikey  Same :/ +  
arkmoses  smh +  
niboonsh  why is it 50% females tho? +2  
imgdoc  felt like an idiot after i figured out why i got this wrong. +2  
temmy  oh shit! +  
suckitnbme  This isn't exactly right as males can still be born as evidenced by individuals III 6,9,11. This basically an x-linked recessive disease. A carrier mother can still pass her normal X chromosome to a son (50% chance). It's just that the other 50% chance of passing an affected X chromosome results in death of the fetus in utero. Thus all males actually born will not be affected. +2  
makinallkindzofgainz  @suckitnbme, Correct, but if you're a live-born male, you 100% for sure do NOT have the disease, so the chance of a live-born male "being affected" is 0. +3  
spow  @suckitnbme it's not X-linked recessive, otherwise every single son would be affected and therefore have died in utero. It's X-linked dominant +3  
qball  Jail-baited +  
srmtn  correct @spow affected females= X linked Dominant +  


submitted by cantaloupe5(78),
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sTih oen was kciytr tub I tinkh yuo ’covedlu oned ihts eno wuihtot keenldgow fo DNMA eesorrt.cp mtSe oltd yuo ttah atamteglu eictsatva othb oM-DnnAN and DMNA stprorcee ubt ti deacativt ylno MNAnn-oD rorscepte in teh yreal aph.es thTa esnma DAMN rtrcopsee aatcveti faetr AoNMn-nD reprco.ets aTht asmne gnheiosmt wsa yediagnl MDAN crtopeer nitvgicata nda the oyln ewrnas ahtt adme ensse sa het gM ibniitingh NMDA at ertigns en.alttpoi cOen het ellc si deezroapdli by -nMNAnDo rsrceot,ep NMAD eectrosrp nca eb iecatdv.at

hungrybox  I forgot/didn't know this factoid and narrowed it to the correct answer and a wrong answer. Guess which one I chose? +14  
yotsubato  >That means something was delaying NMDA receptor activating and the only answer that made sense as the Mg inhibiting NMDA at resting potential. What makes the fasting gating kinetics choice incorrect then? +5  
imgdoc  NMDA receptors are both voltage gated and ligand gated channels. Glutamate and aspartate are endogenous ligands for this receptor. Binding of one of the ligands is required to open the channel thus it exhibits characteristics of a ligand channel. If Em (membrane potential) is more negative than -70 mV, binding of the ligand does NOT open the channel (Mg2+ block on the NMDA receptor). IF Em is less negative than -70 mV binding of the ligand opens the channel (even though no Mg2+ block at this Em, channel will not open without ligand binding. Out of the answer choices only NMDA receptors blocked by Mg2+ makes sense. Hope this helps. +6  
divya  sweet explanation imgdoc +  
lovebug  really~~~ sweet. thankyou :) +  


submitted by imgdoc(135),
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I kithn tsi teh tgrhi LMF r(aae C,) ont the trhig nbsacdue lsenucu ahtt is oedesnil on het rocss osnc.eti fI het seabcndu lecsuun eerw edlsieno seh lwtund'o aehv adoicnubt in hre ltef .eye ehT MFL itmadese crsos tkal tnweebe het cdnsbeua unlecsu on tohb ssdie ot teh LFM no the sopeptio esdi 2( esucbdna ulie,cn 2 FLM oen on aceh dise.) nI her acs,e hre thigr MFL ta'nsw fontucnniig ceenh yhw seh wsa gzigna left utb her irthg dedmli tcsreu 'tansw gnctaonitc ot teaedmi drfalwte gaez.

Tish trepuci eshlp: o2/helpnpcw2.b10pwjan-ptttmu9hg4.teo-utd0hF/mn_hmhw:tso/-a1/oc//pwt/5

imgdoc  *medial rectus +  
quip13  This shows the MLF pathway in a saggital cut: https://ars.els-cdn.com/content/image/3-s2.0-B9781437719260100128-f12-08-9781437719260.jpg +