I ibeeelv tihs smte aym eb ginifrner NEM 1 oysnedrm - isospbel mosgtnriaa iectp(p lrcue ssadeie )xd, hadriopryat ,ndeoaam dna auriitpty amneado guinacs( .)DHAIS t,uB you to'dn need ot evne kwno isth ot gte hist itgrh - ustj sgnika the ecetff fo ihhg HTP no hte emtssy - ceasus iaedrsenc osocstaetl cvattiyi sa wlel sa ariotnmta.u
nca eonemso liapxne wyh it says eh hsa na ttn''cai HPT iorn.tance..tsinco it ot let us kwon atht eht PTH ttionocanecnr is a stuler fo gadty?aonol hp wath's hsi d?x !hsntak
eTh pocectn bgien esetdt si th"aw odes PTH do ttah salde ot pa lm"ahceryicem.c/dpie.bntK:sV.ghot/3sibijg//Ppa
nI iitdnoad ot HTP = loetosasct vityiatc = siedacren l,aimucc ihst rpsoen ludco also be ihxnbigiet tospmmsy fo 1E.NM
tis iprmyra ipmyiryoahtpdsarrhe asedcu by taorahdpyir .nmaoaed ddnotaii of het ctppie rcule ssguetg leZgnlior g&t;--- N1ME
tbu oenn fo tath is mpi cabesue hstt'a not wath ethy ear gainks. All they are knasgi fro si eht ntocifun fo PH.T
Deso eanony wkon eht ingaiscecifn fo ootyemcn ?itiylmto
What helped me to answer this one quite easily was the following rationale:
Hypercalcemia + high PTH -> "primary hyperparathyroidism"
How do high PTH lead to hypercalcemia? Increasing osteoclast activity!
MEN 1 - P P P . Parathyroid, pancreas(gastrin in this case), pituitary lesion. not key to answering this question.
pt comes with symptoms of hypercalcemia, moans, groans, thrones (peeing), psych overtones.
Look at pth and calcium. we see high ca and high pth. We know that high calcium would lower pth...this means the high caclium was due to the high pth (i.e. parathyroid adenoma)
Question is actually asking: what are the effects of PTH? a, c, opposite of PTH
D and E i believed aren't related to PTH
B-Sounds something like the pathogenesis of pseudohyperparathyoidism
F- we definitely know that PTH increases Ca , so by increasing osteoclast maturation/activity you liberate calcium into the blood. this is the best answer.