to snoo-finity ... and beyond!
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Contributor score: 5
ugh, bullshit. I was trying to figure out an actual disease process here.
its primary hyperparathyroidism caused by parathyroid adenoma. addition of the peptic ulcer suggest Zollinger ---> MEN1
but none of that is imp because that's not what they are asking. All they are asking for is the function of PTH.
in a per-protocol analysis, only patients who complete the entire clinical trial according to the protocol are counted towards the final results
"In an ITT population, none of the patients are excluded and the patients are analyzed according to the randomization scheme."
And the sixth D: Diarrhea
a) CAT1 and CAT2 are important enzymes of fatty acid beta oxidation. Glucagon upregulates this process
b) glycogen formation is inhibited by glucagon, so that glucose can be used by cells
c) acetyl CoA carboxylase is used for fatty acid synthesis and thus decreased by increased glucagon
d) glucagon increases ketogenesis
Basically, they want to know what is the function of glucagon.