justanotherimgDoes this mean that in early stages, sympathetic stimulation hasn't happened yet, that is why pulse is weak ? Because otherwise pulse should be rapid (tachycardia in response to sympathetic stimulation caused by decreased stroke volume). +1
chaosawaitsThere will be both tachycardia and weak pulse in early hypovolemic shock+
makinallkindzofgainzThis is not an ambiguous question. It makes perfect sense.+7
khalebIn early shock you have increased SVR due to vasoconstriction. This would cause increased flow to the kidney. I could be wrong but I think what makes that answer incorrect is NOT that RAAS hasn't been activated yet. It is what is causing vasoconstriction via Angiotensin II. What is possibly wrong about that answer is that it says via sympathetic stimulation. I do think it is a little vague between those two answers though. Because you can get sympathetic activation of the RAAS system causing vasoconstriction and blood shunted to vital organs such as the kidneys. Bottom line is you can't argue with weak pulse during hypovolemic shock.... so an obvious right answer. You could make a case for the increased blood flow to the kidney though.+3
zqatan@makinallkindzofgainz no need to dismiss the question... so pretentious +3
chaosawaits@zqatan, interesting, I thought OP was the pretentious one: to each their own, I suppose+
leap1608FA2020 PAGE 310 simply focusing on term hemorrhagic shock preload decreases highly which would decrease the stroke volume. early sympathetic system would only increase the heart rate( as cardiac output= stroke vol*heart rate )+
submitted by โdr.xx(176)
Hypovolemia is a direct loss of effective circulating blood volume leading to:
https://en.wikipedia.org/wiki/Shock_(circulatory)