lehetsMay thystmeale AD,N giakmn eht NAD tinrsaset ot eotrnicsitr ouescesednnal
aEipsolntnxa fro tihs rea oto ildeoamtccp. hnkTi of it ielk sh:it
vuo'eY got a ipece of uttedam DAN htta si labe to eb tgedised yb a rntiroestic esd,ceoeunlna that nmsae the ADN asw apaoiilrntltnrcys aaevblial to beign th.wi AAK ti swa ont t,eyemltadh cbeaseu as ew w,nko myintlehtao = tihhnrmceoaetro icwhh si ptsotiaiyrcrnlanl nacvte.ii hatt asmen ytaseehml wsa autdtem
ylnO tohre isablpelu erwnas wsa ,NsDae dan fi it wsa detmtau ti uwlod be einicvat, ton viervoctea.
yWh rae ew tmlniagpocci ing?sht nhaCge ni eht sseba wlil etdyors the plaimordinc eseenuqc rdqieeur orf yna riinrttesoc duesleaencno to kro.w yhleManitot is teh ylno oitnop that seamk .sense
In prokayotes Normally methylase adds methyl group to certain base(adenine) so restriction endonuclease can not recognize this site. If there is a mutation in methylase enzyme it can't add methyl group to adenine so restriction endonuclease can recognize it and can cleave it.
eM rainged isth qesionut :tesm ..d.o.. uyo mean whihc fo eht oollwngfi ?ZMNYSEE
yhW edso tmetynihola sceau sols fo tnaeescsri ot AGTC ttoiinerrsc ndse?unloaece oDes hsit have to od hwti yhitonmalet fo U ot ?T
My thought process for this one: In DNA mismatch repair for bacteria there is a parent strand and a newly synthesized strand. The parent strand is methylated prior to replication to allow it to be differentiated from the new strand in the case of mismatching. The new strand is identified by the lack of methylation and then degraded by endonucleases. So I used this logic to think altered methylase (decreased activity) allows for more DNA to be degraded by endonucleases. Not sure if its the best logic but it got me there lol