A bit confused here because I thought T4 is converted to T3 INSIDE peripheral tissue. Why would serum T3 levels rise then?
According to this article, patients with hypothyroidism taking too much levothyroxine ended up having NORMAL serum T3 levels: https://pubmed.ncbi.nlm.nih.gov/26738396/#:~:text=In%20levothyroxine%20(T4)%2Dinduced,in%20over%2Dtreated%20hypothyroid%20patients.
Anterior lead MI leading to cardiogenic shock with decreased LV contractility.
SVR rises in cardiogenic shock as compensatory response to low CO. PCWP rises because in anterior MI there's decreased contractility and blood backs up into LA.
That leaves one answer option of high SVR and high PCWP and you don't even have to think about PVR ;)
GnRH agonists like Leuprolide are effective for patients with breast CA because if given in a continuous fashion, they downregulate the GnRH receptor in the pituitary and ultimately decrease FSH and LH.
The key for this being dissecting aneurysm is that it's a widened aortic arch. Also, there's uneven pulses ("diminished pulses in the left upper extremity") meaning that blood is not passing from the aortic arch to the left subclavian artery (it's not passing the aortic arch).
When I read "ginkgo biloba" as my answer choice (E) I thought I was getting delirious lmao