Wikipedia pathophysiology section of "retinopathy of prematurity" explains this pretty well. Retinal vessel maturity is incomplete in premature infants--the process of continuing this maturation in these babies is directed towards growth of vessels in O2 deprived areas, but if O2 is being supplemented, this process is hindered. Therefore when O2 supplementation is removed, excessive vessel proliferation going into the vitreous humor (not where it is supposed to be) leads to retinopathy of prematurity (defined as "disorganized growth of retinal blood vessels leading to scarring and retinal detachment")
ion"rhcc hhgi engoyx uirtaosants can dalervyes aftecf glnu dan eey meutoocs fo terempr ts"fin .nanikl
rgaee htwi a' eryeuh.rbtk itnygr to asy atth ryapimr aglo fo tenmetatr itwh tpaspmneuell 2o is ot ekam rues hreet si no ilratne adm.aeg
hyw is tcinpreoot fo the drhcioo p,sulex mofr tauvnrncreitairl ureuprt enasyocrd to irealmng mxtiar eghmoerhra ton a iesblops sraw?en
submitted by โhyoscyamine(59)
It can be re-accessed by making a purchase.
Purchase your membership here
AF pg 1.72 oTo mcuh eonityxgona nca caues eref clairad emagda egaldin to hpyetaoirnt fo earrmytptiu