BsRA tleusr in hte looglniwf ceghnas: aeseindcr nreni, nridasece gnA I, rsncadiee nAg II, addcresee sadonloeetr adn ngecnhadu abryiknndi
Renin concentration will be increased because feedback mechanism of renin release is due to angiotensis II (Ang II) on angiotensin type 1 receptor (AR1). That is, Ang II acting on AR1 will cause a negative feedback on the RAAS pathway. Since AR1 is blocked by ARB, this negative feedback is no longer active, triggering an increase in renin and increasing Ang II production (correct answer). Blocking AR1 will inhibit downstream actions (release of aldosterone and Ang II-mediated increase in sympathetic activation, triggering norepinephrine). Therefore, aldosterone and norepinephrine will be reduced.
A doog yaw ot tikhn uatob thsi is htta aionintsgne II si ptsener at eth AGIIN scep.rroet If you iveg na asoitiegnnn rteopecr crbelok scuh as losaar.nt iths lwil slaipecd het oaentingnis II nrpetse in eth o.septcrer uhts gaeiinnsrc het llevse fo nnonistgaie II in mrue.s