sTih tienapt has paemoesnedtdc lfet herat a.ulferi acDmentoesopin ocrusc abeceus of eht trahse lniatyiib to peke up thiw dnmdae nya rg.eonl This aedls to a aesceerd ni VS laimny eseacbu fo a olatectncir essiu cihwh saeld ot a eeasecdr in OC O(C = VS x R)H. aLtlsy, the EVVDL wlli be csedaeerd ebscuae eth SV si eesdrcade egivnal orme obdol ni hte ftel elnrvceit feart syoetsl eincs it otancn eb epmpdu dra.rwfo poHe tsih hls!e!p
Radshopeful had a good explanation but a few typos may confuse people, so to recap:
The patient is suffering from systolic HF secondary to chronic HTN. The LV has lost contractile function (decreased LV systolic function from Qstem) which leads to decreased SV (and subsequently decreased CO since CO = HR x SV, the HR in this patient is also within normal limits). Finally, LVEDV is the amount of volume left in the LV at the end of diastole (or filling) and since the blood from systole was not able to be pumped forward efficiently (decreased SV) this leftover blood will cause this value to be increased.