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If the tdoorocnh ifdeal ot pveoedl neht teh neietr CNS uoldw tno oevdple sa hte rthooodcn duscine miaroonft fo enaurl l.peta
If hte lareun betu efladi to dpvleeo hnte teh oelhw CNS odwlu ont eahv .edpveodle
olkY sca si reietlarnv to shti .tnepati
nheW erluna ecsrt cell ti has etiffnrde utcsmeoo ni enfdirtfe ises.sut lerFaui of nrueal stecr ot amteigr ni ahetr nac uasec Tipssoinnraot of reatg el,vssse gToylerta fo ltlFao, or etsriePstn unsrtcu reraousits. ruiFlae of nurael srestc to tgmarie ni IG nac ecuas iphrsucsHrgn esiasde cateln(gino .eloo)angcm rceaehTr lolCsin dSynomer acn cruco ehnw nalreu srtce elcls flai ot atrigme iton t1s eagrlnyahp rc.ah ralNeu tube cesfted sha thigonn to od iwth euiralf fo unreal estcr riminaogt hugoht.
Paraxial mesoderm means mesoderm surrounding the axis, aka the neural tube. They eventually form a pair of somites on either side. Somites then differentiate into:
In this case, the patient is lacking spinous processes on top of having spina bifida. Maybe you were thinking spina bifida = neuropore defect, but what does this have to do with sclerotomes? This is what I found:
"Failure of closure of the caudal neuropore during embryogenesis will lead to spina bifida. This condition is always marked by a local lack of osteogenesis, or bone growth. The reason is that the correct differentiation and placement of underlying tissues (the spinal chord and spinal nerves and associated tissues) induce the osteogenesis, even small flaws in the underlying tissues can lead to a problem in the formation of the spine (vertebrae)." Source
So I'm guessing that when the question asks for "cause of the defect" they are talking about the vertebrae, not exactly the cause of spina bifida, which would be failure of neuropores to fuse. If that was an answer choice then def pick that, but the lack of vertebrae is due to lack of sclerotome fusion (because spina bifida could not induce it properly).