Wikipedia pathophysiology section of "retinopathy of prematurity" explains this pretty well. Retinal vessel maturity is incomplete in premature infants--the process of continuing this maturation in these babies is directed towards growth of vessels in O2 deprived areas, but if O2 is being supplemented, this process is hindered. Therefore when O2 supplementation is removed, excessive vessel proliferation going into the vitreous humor (not where it is supposed to be) leads to retinopathy of prematurity (defined as "disorganized growth of retinal blood vessels leading to scarring and retinal detachment")
c"orcinh high xgyneo taitassruon nca rsdeelyva feaftc guln dan yee msecootu fo ptererm snt".i nfaknil
eegra hwti ar.yetk hrb'eu igytnr ot asy atth mpairry ogla fo tremteatn itwh tpuaslneemlp o2 is to keam user hrtee si no ritlane de.mgaa
why si coienptotr fo eht ohrodic xpulse, frmo rltarvaciurtienn utrperu osrdceyan ot gamleirn ixmtar rhhemoareg not a pilbseos aens?rw
submitted by โhyoscyamine(59)
It can be re-accessed by making a purchase.
Purchase your membership here
$279$49FA pg .271 Too mhcu xnioyteoang anc uasec rfee dilcaar amaedg landieg ot tnerohiaytp fo yeumprirtat
$279$49