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NBME 17 Answers

nbme17/Block 1/Question#38 (reveal difficulty score)
A 69-year-old woman comes to the physician ...
Area marked "G" ๐Ÿ” / ๐Ÿ“บ / ๐ŸŒณ / ๐Ÿ“–
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submitted by โˆ—bingcentipede(356)
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https://imgur.com/RQGrWLw

G represents the primary somatosensory area of the parietal lobe. The stem describes a 69 (nice) year old woman with sensory issues on the left side. She presents w/ a Babinski sign on the left, decreased somatic sensation in the left foot, agraphesthesia (when you "draw" a number on someone's skin and they can't interpret it) on the plantar surfaces of the toes, decreased position sense in the toes. The question says there is an edematous area in the cerebral cortex of the right hemisphere.

I had trouble with this, but I think it's describing somatosensory because of the sensory problems. Don't understand the UMN lesion (Babinski). Here's what Wikipedia says: "Lesions affecting the primary somatosensory cortex produce characteristic symptoms including: agraphesthesia, astereognosia, hemihypesthesia, and loss of vibration, proprioception and fine touch (because the third-order neuron of the medial-lemniscal pathway cannot synapse in the cortex). It can also produce hemineglect, if it affects the non-dominant hemisphere. Destruction of brodmann area 3, 1, and 2 results in contralateral hemihypesthesia and astereognosis."

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mittelschmerz  The Babinski tripped me up too, here's what I found: "Sixty percent of the CST fibers originate from the primary motor cortex, premotor areas, and supplementary motor areas. The remainder originates from primary sensory areas, the parietal cortex, and the operculum. Damage anywhere along the CST can result in the presence of a Babinski sign." from https://www.ncbi.nlm.nih.gov/books/NBK519009/ +18
yourmomsbartholincyst  I think this question is more simply about the topographical arrangement of the homunculus (which I once again somehow managed to flip backwards during the exam). Lower extremity is topographically mapped to more medial portions of the somatosensory cortex, hence letter G and why ACA strokes tend to affect the LE more. Homunculus, our favorite hunk, FA2020 pg 502 +17
j44n  youre occluding the MCA blow flow from the MCA flows front to back so if you occlude the section that provides blood flow to the precentral gyrus you will also occlude the section that supplies the sensory portion http://what-when-how.com/neuroscience/blood-supply-of-the-central-nervous-system-gross-anatomy-of-the-brain-part-1/ +
j44n  excuse me i miss spoke its the ACA because its her legs, look at figure 4-2 in the link on my previous comment +
j44n  so if you have motor and sensory the infarct is in the portion that belongs to the motor portion, if you have sensory only youve occluded the artery more distally +
jy1544  I thought G was indicating the primary motor cortex in the frontal lobe from a medial view (anterior to the central sulcus), hence the weakness - and the sensory disturbances were due to edema affecting the adjacent somatosensory cortex https://web.duke.edu/brain/siteParts/images/fig1-5_2018.jpg +9
juhee  From medial sagittal view of brain, the sulcus between G and H was marginal sulcus (not central sulcus). It is extension of cingulate sulcus. And it is an excellent landmark to help confirm the location of the central sulcus, which is located one sulcus anterior to the marginal sulcus. So G is indicating the primary somatosensory cortex. Here's the image. https://prod-images-static.radiopaedia.org/images/30139451/e006b3135364e41e213e83fc2a99ac_gallery.jpeg +
chaosawaits  This is purely a homunculus question. Any further thought into it is just overkill. +



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submitted by sschulz2013(7)
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Pre-central gyrus= motor deficit (muscle weakness) UMN lesions (cerebrum) will cause increased reflexes (I think of Babinski present as hyperreflexia) IDK if it's like seizures, but I know seizures can start out as primary motor and expand to involve the sensory cortex as well. Maybe it was expansion of the edematous area

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j44n  i agree you cant have UMN signs unless the precentral gyrus is infarcted +
smitt1  Is A just the sensory cortex and therefore wouldn't explain the UMN/Babinksi? +
chaosawaits  I have A as lateral primary somatosensory cortex, B as lateral motor cortex, C as prefrontal cortex, D as superior temporal gyrus, E as supramarginal gyrus, F as medial frontal lobe, G as medial primary somatosensory cortex, H as parietal lobe and I as occipital. +
rwd1027  Jไปฃ่กจไป€ไนˆ๏ผŸ +
burak  ๅฎƒไปฃ่กจไบ†ๆกฅ +



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