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j44n
i agree you cant have UMN signs unless the precentral gyrus is infarcted
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smitt1
Is A just the sensory cortex and therefore wouldn't explain the UMN/Babinksi?
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chaosawaits
I have A as lateral primary somatosensory cortex, B as lateral motor cortex, C as prefrontal cortex, D as superior temporal gyrus, E as supramarginal gyrus, F as medial frontal lobe, G as medial primary somatosensory cortex, H as parietal lobe and I as occipital.
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Pre-central gyrus= motor deficit (muscle weakness) UMN lesions (cerebrum) will cause increased reflexes (I think of Babinski present as hyperreflexia) IDK if it's like seizures, but I know seizures can start out as primary motor and expand to involve the sensory cortex as well. Maybe it was expansion of the edematous area