mass in the esophagus with solid/liquid dysphagia. I'm not sure what the histo shows but on the bottom there are more flat cells and the top there are wider spaces within the cells with some fat, so I think it maybe a healing ulcer leading to a stricture.
anways other pathogens are more associated with other infections
A) gallstones and cholangiocarcinoma B) esophagitis c) liver abscess E) i think miliary TB can present in the liver
mass in the esophagus with solid/liquid dysphagia. I'm not sure what the histo shows but on the bottom there are more flat cells and the top there are wider spaces within the cells with some fat, so I think it maybe a healing ulcer leading to a stricture.
anways other pathogens are more associated with other infections
A) gallstones and cholangiocarcinoma B) esophagitis c) liver abscess E) i think miliary TB can present in the liver
My understanding is that H. pylori actually decreases the risk for esophageal disorders (ex. adenocarcinoma)
How would a chronic H. pylori infection lead to healing ulcer/ stricture formation or adenocarcinoma?
How does that picture help at all? Is it just for ruling out or can you rule in H. pylori with it?
I feel like the best strategy for this question is ruling out everything else. The amount of information that definitively says H. pylori, to me, is minimal.
Clonorchis: is a liver fluke, causes problems with gallbladder CMV: usually in immunocompromised patients, no giant cells on histology Entamoeba: liver abscesses TB: no lung involvement, really no indications of TB at all, honestly
submitted by โprosopagnosia(5)
GERD --> acid damages mucosa --> knockout of the mucosa leading to decreased stem cells --> ulcers form and heal via fibrosis --> stricture --> dysphagia. Complication of this chronically would be Barrett Esophagus? (I can't tell if those are cells filled with fat or goblet cells that replaced the squamous epithelium). Barrett Esophagus can progress to Adenocarcinoma of the esophagus. Source: Pathoma pg 102