In idbeicta to,eiosscdaik ehret is eaeridnsc aidc ni hte calateurrllxe acse.p Acgdnrioc to FA eehtr is a rsaaelrtllncu itfhs ued to edercased iunnisl tath ledas to eomr H+ entrnieg teh lelc in cnaghexe orf +.K hiTs edlsa ot lrphyiaeemak whit deltepde lcrnreltaiaul sosrte fo .K+ rTeeh si osal ciosotm seirsuid that adsle to csedanier +K lsos in hte nreui nda ttalo ydbo +K inodpl.eet eTh oiuentsq skas ttah is otms yekill ot eaercdes hiwt nuliisn eyhatp:r rsuem ismpatous ncotcrneaitno illw edcarese sa +K si onw aenhegdcx rfo H+ idnies het cl.el
This explanation is faulty. Insulin causes K to go inside via upregulating NaKATPase activity. Nothing to with H+ exchange as far as Insulin is concerned.
H and K exchange is separate from this.
I gaere ahtt muerS K dercaees is teh teretb ,wrsena but od'unwtl Uiern K aslo daersece ecno lobdo grusa gose odwn hiwt nisilun mrneattet e(.i. oimcost sdisiure udwlo lne?)ses
submitted by โan_improved_me(91)
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iIsnunl dsoe ylap a role ni gbgiirnn back K+ noti ellsc via nluaodmoti fo KH+/+ cx.haneeg shTi is ucaeseb ti elhps ot saersdd teh accoieotdkti estat of eth tnti.epa By nbrngigi Hp cselro ot ,ebinslae rheet is ruertn fo +K noit eht c,lles hweli H+ soecm akcb uot fo eth lc.lse
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