This is describing hereditary hemorrhagic telangiectasia (FA 2019 p. 312). It is also known as Osler-Weber-Rendu syndrome. It is an autosomal dominant disorder of blood vessels. Findings include blanching lesions (telangiectasias) on skin and mucous membranes, recurrent epistaxis, skin discolorations, AV malformations, GI bleeding, and hematuria.

you cant blame anyone for your being late. You have to deflect and stay broad because it creates a distrust in the medical system all the other ones place blame else where or make you look like the encounter isnt a priority

This protein is an uncoupling agent (likely thermogenin which is found in brown adipose and serves to increase heat production. Uncoupling agents will cause increased permeability of the mitochondria membrane, necessitating more oxygen consumption in order to generate the same ion gradient. Since more oxygen is consumed to generate the same gradient, more oxygen would be consumed to generate the same amount of ATP, so the ratio of oxygen consumption to ATP generation increases. (FA2020 p78)

Another way of thinking about this is that oxygen will continued to be consumed to attempt to generate the gradient, but the leaky membrane will prevent the gradient from being formed properly and therefore prevent ATP from being formed. Thus there is the same amount of oxygen consumed for little to no ATP being made so again the ratio of oxygen consumption to ATP generation increases. This is closer to what actually happens in the body but I found it easier to think about it the first way.