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Retired NBME 25 Answers

nbme25/Block 2/Question#8 (reveal difficulty score)
The breakdown of dipeptides and tripeptides ...
Intestinal mucosa ๐Ÿ” / ๐Ÿ“บ / ๐ŸŒณ / ๐Ÿ“–
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submitted by โˆ—shak360(23)
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ediespDtip nad eiipdsterpt aer dduopcre by eth ciytivta of rptsnyi nad prhyiomncyts hintiw hte uelnm of hte emdo.udnu eFer noaim cisda rea cdrpuoed opnu rufreht deoitgsni of teshe llams depteisp wihtni the bhusr drebor fo het iieatlsnnt mo.cuas

eTh mulen of hte ndudmuoe is gorwn sabeuce hatt si weher graler oxlcpem potdyeplsipe rea orkenb wdon ot di- dna dpetrseitpi btu the tiaonc fo kiagnt -id nda tptrepdisie dnow ot ndlivuiiad aimon csdai peashnp ni the ntasniltei .uomsca

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Maybe a way to remember this would be to think about Hartnup disease, an AR deficiency of neutral amino acid (primarily tryptophan is implicated) transporters in the proximal renal tubular cells and on ENTEROCYTES. This leads to neutral aminoaciduria and decreased absorption from the gut and little breakdown of dipeptides and tripeptides to amino acids in the intestinal mucosa. Decreased absorption from the gut causes decreased tryptophan for conversion to niacin and pellagra-like symptoms (diarrhea, dementia and hallucinations, and dermatitis in the C3-4 circumferential "broad collar" or "Casal necklace" dermatome, hyperpigmentation of sun-exposed limbs).

So if you can't convert dipeptides and tripeptides to amino acids in the ENTEROCYTES in the INTESTINAL MUCOSA, then you can get Hartnup disease.

+1/- shak360(23)

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