When acetylcholine binds postsynaptic nicotinic acetylcholine receptors, each receptor causes Its own postsynaptic voltage change, which is called an end-plate potential.
The summation of the end-plate potentials represents the membrane potential.
The synaptic acetylcholine concentration influences the amplitude of end-plate potentials
Blocking presynaptic, VG Ca channels would most decrease the end-plate potential amplitude!
submitted by ∗solidshake(25)
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$279$49