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Comments ...
skilledboyb
Paralysis is a finding common both the UMN and LMN. The distinguishing findings are things like:
atrophy (LMN): 30 minute onset not long enough to see this
reflex changes, spasticity, etc: not mentioned in the stem
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fatboyslim
I chose G too thinking it is the ventral horn but G is the anterior spinothalamic tract. To answer your UMN vs LMN question, maybe it's because in acute spinal trauma you get a spinal shock which leads to flaccid paralysis, even if the lesion is supposed to cause UMN sx (like in this case affecting corticospinal tract). And then with time, the patient will develop UMN lesion sx (e.g. spastic paralysis, spasticity, etc.)
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fatboyslim
^Correction: G is the lateral spinothalamic
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Subcomments ...
I understand that damage to the area labeled E would cause issues with the corticospinal tract. However, wouldnt this damage cause UMN findings as it is before the transition to LMN in the anterior horn? In the stem we read that the pnt has LMN finding of "unable to move" So why are we seeing LMN and not UMN findings? thanks in advance