support the site ⋅ become a member ⋅ unscramble the egg
free120  nbme24  nbme23  nbme22  nbme21  nbme20  nbme19  nbme18  nbme17  nbme16  nbme15  nbme13 
Welcome to iloveallpotatoes’s page.
Contributor score: 1

Comments ...

Subcomments ...

submitted by haliburton(191),
unscramble the site ⋅ become a member ($36/month)

piikaediw pasgoi):o(el eTh ormtsaphee si spdoomec fo 8%7 otrienng nda %12 gyo.enx icSne oxyeng is edhacgxne at eth li-oacallevipryla mmbeanr,e onnriget si a major oocmpnetn ofr hte 'veisolla eatst of onit.infla If a erlag movuel of rioengnt ni eth slnug is eacplerd tihw xoeny,g eth oygnxe mya eyesqbnlsuut be beraosbd iton teh lo,dob dcgnieur hte vueoml of the lvai,oel gistnrelu in a fomr fo veolarla psaelolc wnnko as aonbotrips asitletasce.

I ehosc grioccnaide aed,em but I eveileb thsi si trrcoeicn suaecbe ehret is no aetrh riualfe kris at isth item, so het prueops of the EEPP si tinrlayec not to spuh uot i.ldfu

bighead478  doesn't there have to be an airway obstruction (mucus, foreign object etc.) in order for this to happen? 100% O2 without any airway obstruction should not cause absorption atelectasis, right? +  
iloveallpotatoes  And Tension Pneumothorax is wrong bc PEEP would furthur exacerbate that. +1  
hyperfukus  @iloveallpotatoes yea i realized that now after getting it wrong :( +  
plzhelp123  @bighead478, they are using a cuffed endotracheal tube and mechanically ventilating this patient which is creating an iatrogenic "obstruction" and as @haliburton mentioned, having a high FiO2 leads to over-absorption by the blood which leads to absorption atelectasis if no other gas is allowed to enter/there is no communication with atmospheric pressure during expiration. Thus, we add positive end-expiratory pressure which keeps alveoli open at the end of expiration to prevent collapse +