vitals and need to be treated first, so atropine and then pralidoxime.
important to note that organophosphates become irreversible at a certain point so pralidoxime does still need to be given early
mass in the esophagus with solid/liquid dysphagia.
I'm not sure what the histo shows but on the bottom there are more flat cells and the top there are wider spaces within the cells with some fat, so I think it maybe a healing ulcer leading to a stricture.
anways other pathogens are more associated with other infections
A) gallstones and cholangiocarcinoma
c) liver abscess
E) i think miliary TB can present in the liver
He has a down and out pupil, caused by CN III palsy. His gaze is due to unopposed action of the lateral rectus and superior oblique; ptosis due to denervation of levator palpebrae superiosis.
The only injury listed that could cause a CN III palsy is aneurysm of the PCA compressing the ocularmotor nucleus.
Strep Viridans is oral flora, causes subacute endocarditis affecting previously damaged valves, and is often associated with sequelae of dental procedure.
A) enterococci can also cause subacute endocarditis but they are gamma hemolysis and follow GI/GU procedure
B) beta hemolysis and causes acute
c) beta hemolysis and causes acute
e) alpha hemolysis but unlikely to cause endocarditis, causes meningitis, otitis, pneumonia, sinusitis - (MOPS)
TLC decreased suggests restrictive disease. Reticular pattern suggests pulmonary fibrosis
She has motor and sensory symptoms. This r/o both kinds of simple. Her seizures are not described as tonic/clonic movements, and include periods of impaired consciousness. This rules out generalized tonic/clonic. There is a post-ictal state, that rules out absence.
Also the lip smacking is characteristic of automatism, which is found in complex partial seiuzres
Acute MI and mitral regurg (from the murmur) leads to LV failure and backflow of blood into the lungs.
This leads to increased pulmonary hydrostatic capillary pressure. This will lead to excess volume leaking from the pulmonary capillaries into the interstitial and this will manifest as pulmonary edema (crackles).
Pulmonary edema will interfere with gas exchange leading to hypoxemia.