so, the Key words that no one is mentioning : communicating hydrocephalus

the pathophys goes like this :

an inflammatory setting (i.e., subarachnoid hemorrhage) yield fibrosis / scarring of the arachnoid granulations => impaired CSF drainage

the key points / concepts they are trying to test here : 1. do you know what communicating hydrocephalus (without them telling you those words) 2. do you know what's the pathophysiology (of communicating hydrocephalus) is ?

Normally the arachnoid villi drains the CSF from the subarachnoid space to the venous system; if this part becomes defective then you can imagine all that CSF now building up in the subarachnoid space.

The patient has a leaking berry aneurysm --> this leads to a subarachnoid hemorrhage (or leakage into subarachnoid space) --> acute SAH can lead to decreased absorption and movement via arachnoid villi

simply the blood inside the CSF become blocked through arachnoid granulations ==> decrease absorption of CSF==>communicating hydrocephalus.

could it also be possible to get this by remembering the different ventricle spaces as well. i honestly didnt remember what hydrocephalus this could be. But i remembered the mnemontic LIT AFF to SAD.(Lateral ventrical, intraventricular foramina of monroe, third ventrical, cerebral aqueduct, fourth ventricle, foramina of lushka, subarachannoid, arachanoid granulations, dural venous sinus). And i recognized they were talking about a widing of subarachanoid space and thought what was the closest thing to interefere and thats how i got to B.

Also i just want to say looking at the answers i know its a communicating hydrocephalus, i just wanna know if this was also a good way to extract the answer or if i just winded up getting lucky a little LOL.