Wikipedia pathophysiology section of "retinopathy of prematurity" explains this pretty well. Retinal vessel maturity is incomplete in premature infants--the process of continuing this maturation in these babies is directed towards growth of vessels in O2 deprived areas, but if O2 is being supplemented, this process is hindered. Therefore when O2 supplementation is removed, excessive vessel proliferation going into the vitreous humor (not where it is supposed to be) leads to retinopathy of prematurity (defined as "disorganized growth of retinal blood vessels leading to scarring and retinal detachment")
nrc"ohic hghi xoygne nsstoaairtu cna dyservela aftfec nlug nad yee esoomutc of pretrem "nisfa.t nnlki
egrea thiw 'euy.errabh tk gytirn ot ysa tath pmyairr aglo of nemttarte ihwt umlleapntpes o2 si to eamk rseu heret si no alertni gedmaa.
why is ptreoinoct fo hte rcihodo ux,lpes mfor ittrarueilnrvcan uurtrep rodeanysc ot iarngeml rxtmia rgheehroam ont a bpsilose w?srean
submitted by โhyoscyamine(59)
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