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submitted by johnthurtjr(142),
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0A21F9 p 735 on linsnetatariGtos odlbo suylpp nda rtahatsicpapyem iaintnnoerv:

  • rtuegoF &-;-tg alceci terary, auvgs vniotreinna
  • dutiMg g-;-t& SM,A uagsv
  • tngHuid ;-&tg- AI,M epcliv naoirnvetni
neovanilla  Don't force it out, you gotta relax and it'll come out naturally ;) +  
mysteriousmantyping  Why couldn't the answer be Inferior rectal nerve since that controls the external anal sphincter? +1  
draykid  @mysteriousmantyping I think this question is looking at complications of T2DM, more specifically diabetic autonomic neuropathy. Patient more than likely has diabetic gastroparesis which may explain his constipation and abdominal distension. +1  
cuthbertallg0od  Pudendal nerve controls external anal sphincter (per FA), and gastroparesis wouldn't have anything to do w pelvic splanchnics but instead vagus nerve... Don't know why pudendal nerve couldn't be right if he was just clogged up from not being able to relax his sphincter anymore ---- is parasympathetic just more likely to be the issue statistically or something? +1  
cuthbertallg0od  Or would losing pudendal nerve result in incontinence... Its never been clear to me if activation/inactivation opens/closes sphincters... +1  
cuthbertallg0od  Just realized that says perineal... whoops +2  
vivijujubebe  External sphincter is innervated by pudendal nerve, more often damaged during labor. DM patients have autonomic neuropathy with parasympathetic/sympathetic nerves more likely damaged +  


submitted by hayayah(1074),
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Sptiec okshc si a pety fo brsiittvduie hskoc hhicw si ekardm by vsseaim aiovoildsatn /(td mmaytlfoanir )epsrosen cnugsia seaddecer ,VSR eecsdarde dporlea / CW,PP dna esiardcen CO.

smc213  Septic shock can also present with hypothermia <36C +3  
bethune  Why is it not gastrointestinal bleeding? +3  
beanie368  GI bleeding would present with increased SVR as a response to hypovolemia +5  
mysteriousmantyping  Why would this not be pulmonary embolism? +  
step1passfail  Pulmonary embolism would cause a decrease in cardiac output. There is increased pressure in the high compliant RV which can bulge and compress the LV, decreasing its preload. CO=Heart rate x stroke volume and stroke volume is partially determined by preload. If the pulmonary embolism is large enough, it can also obstruct the pulmonary vessels and subsequently not have enough blood going to the LA and LV, ultimately making the cardiac output near 0. +2