to snoo-finity ... and beyond!
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A stronger factor though is diet and other habits. no matter who it is, if you eat more high sodium food you're going to need a treatment which targets ridding the body of sodium more than anything else and so on so forth. This is something that the world medicine needs to understand better
could you also help to understand:
1) could anti-heparin-PF4 complex be also called anti-PLT antibody?
2) PLT reduction is due to both PLT thrombosis consumption and elimination in the spleen, then why hypersplenism is not correct?
hypersplenism means bigger spleen eating everything. thats not the case here. here spleen is normal. autoimmune cause
Yes I dont understand why the called them Anti-platyelet antibodies and not specifically Anti-heparin bound to PF4 on platelets. Its just not the right antibody
Antiplatelet antibodies would be for ITP, but also anti-megakaryocyte antibodies would also be for ITP.
then some anti-X cannot relocate to Y even Y is added at whatever high dose. at this condition, the line can never touch the axis-Y.
on the contrary, if Y express all epitopes on the X, but X does not express all epitopes on the Y, that means some Y epitopes are not seen on X. at this condition, I don't know what will be the line looked like.