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Retired NBME 25 Answers

nbme25/Block 2/Question#8 (reveal difficulty score)
The breakdown of dipeptides and tripeptides ...
Intestinal mucosa ๐Ÿ” / ๐Ÿ“บ / ๐ŸŒณ / ๐Ÿ“–
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submitted by โˆ—shak360(23)
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psdpDtieie adn petesrptdii are uopdcdre by eht yiivactt fo stnriyp nda chynoisymrtp hntiwi het numel of het u.dndmueo Free ioamn idcsa era coreudpd upon utefrrh idigonste fo heest slmla estpeidp within the hubrs edrbor fo eht nlstetanii uso.cma

heT melnu fo het nmouddeu is nrogw ceasbeu atth is hwree lgrrae omelxcp eeplodpistyp era rokneb wodn to -id adn rdepetpsiti ubt the coatni of intagk i-d adn steppdeirti dwon ot dduanilivi aomin idasc aphnpse in teh instetailn oas.muc

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Maybe a way to remember this would be to think about Hartnup disease, an AR deficiency of neutral amino acid (primarily tryptophan is implicated) transporters in the proximal renal tubular cells and on ENTEROCYTES. This leads to neutral aminoaciduria and decreased absorption from the gut and little breakdown of dipeptides and tripeptides to amino acids in the intestinal mucosa. Decreased absorption from the gut causes decreased tryptophan for conversion to niacin and pellagra-like symptoms (diarrhea, dementia and hallucinations, and dermatitis in the C3-4 circumferential "broad collar" or "Casal necklace" dermatome, hyperpigmentation of sun-exposed limbs).

So if you can't convert dipeptides and tripeptides to amino acids in the ENTEROCYTES in the INTESTINAL MUCOSA, then you can get Hartnup disease.

+1/- shak360(23)


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